Kejadian Keloid Menurut Golongan Darah Pada Pasien Pasca Luka Di RSUP H.Adam Malik Medan

KEPUSTAKAAN

Moustafa, MF, Abdel-fattah, MA & Abdel-fattah, DC 1975,”Presumptive evidence of the
effect of pregnancy estrogens on keloid growth”, case report : Plast Reconstr Surg, vol. 56,
pp. 450-453

Mast, BA 1992,”Wound healing : Biochemical and clinical aspect in the skin”,ed IK cohen, B
Diegelmann & WJ and Lindblad WB, Saunder Company, Philadelphia,pp. 344

Nemeth, AJ 1993,”Keloid and hypertropic scars”, J Dermatol Surg Oncol, vol. 19, no. 8, pp.
738-746

Prockop, DJ & Kivirikko, KI 1995,”Collagens : molecular biology, disease, and potentials
for therapy”, Ann Rev Biochem, vol.64. pp. 403-434

Raghaw, R 1994,”The role of etracellular matrix in post imflammatory wound healing and
fibrosis”, FASEB J, vol. 8, no. 11, pp. 823-831

Santoro, MM & Gaudino, G 2005,”Cellular and molecular facets of keratinocytte
reepithelizzation during wound healing”, Exp Cell Res, vol. 304, no. 1, pp. 274-286


Schierle, HP, Scholz, D & Lemperle, G 1997,”Elevated levels of testosteron reseptors in
keloid tissue: an experimental investigation”, Plast Recontr Surg, vol.100, no. 2, pp. 390-395,
discussion 396

Sukla, IM, Arora, NP & Arora, MM 1975,”Corneal keloid”, Indian J Opthalmol, vol. 23,n0.
2, pp. 18-19

Slemp, AE & Kirsner, RE 2006,”Keloid and scars : a review of keloid and scars, their
patthogenesis, risk factors, and management”, Curr Opin Pediatr, vol. 18, no. 4, pp. 396-402
Stadelmann, WK, Digenis, AG & Tobin, GR 1998,”Physiology and healing dynamics of
chronic cuttaneus wound”, Am J Surg, vol. 176, no. 2A Suppl, pp. 26S-28S

Universitas Sumatera Utara

Sylvia, CJ 2003,”The role of neutrophil apoptosis in influencing tissue repair”, J Wound Care
, vol.12, no.1, pp. 13-16

Werner, S & Grose, R 2003,”Regulation of wound healing by growth factors and cytokine”,
Physiol Rev,vol. 83, no. 3, pp. 835-870


Urioste, SS, Arndt, KA & Dover, JS 1999,”Keloids and hypertropic scars : revieww and
treatment strategies”, Semin Cutan Med Surg, vol. 211, no. 2, pp. 159-171

Ong, CT, Khoo, YT, Tan, EK 2007,”Epithelial - mesenchimal interactions in keloid
pathogenesis modulate vascular endothelial growth factors expression and secretion”, J
Pathol, vol. 211, no. 1, pp. 95-108

Louw, L 2007,”The keloid phenomenon : progres toward a solution”, Clin Anat, vol.20, no.
1,, pp. 3-14

Knigton, DR, Hunt, TK 1983,”Oxygen tension regulated the expression of angiogenesis
factors by macrophages”, Science, vol. 221, no. 4617, pp. 1283-1285

Hinz, B 2006,”Masters and servants of the force : the role of matrix adhesions in
myofibroblast force perception and transmission’’, Eur J Cell Biol, vol. 9, no. 3-4, pp. 175181

Ford, LC, King, DF, Lagasse, LD & Newcomer, V 1983,”Increased androgen binding in
keloids : a preliminary communication”, J Dermatol Surg Oncol, vol. 9, no. 7, pp. 545-547

Cheng, JC, Evans, JH, Leung, KS 1984,”Pressure therapy in the treatment of post burn

hypertrophic scars”, Burn Ince Therm Inj. vol. 10, no. 3, pp. 154-163

Bloom, D 1956,”Heredity of keloids : review of the literature and report of a family with
multiple keloids in five generation”, N Y State J Med,vol.56, no. 4, pp. 511-519

Apfelberg, DB, Maser, MR, White, DN & Lash, H 1989,”Failure of carbon dioxide laser
excission of keloids”, Laser Surg Med, vol. 9, no. 4, pp. 382-388

Universitas Sumatera Utara

Appleton, I, Brown, NJ & Willoghby, DA 1996,”Apoptosis, necrosis, and proliferation :
possible implication in the etiology of keloids”, Am J Pathol, vol. 149, no. 5, pp. 1441-1447

Toure, Abas M. Is There an Association between Keloids and Blood Groups. Available in:
ISRDN Dermatology Vol 2012. 2012

Hilmer MP, Macleod SM, 2002. Experimental

keloid


scar

models: A review of

methodological issues journal. Journal of Cutaneous Medicine and Surgery 6(4).

Perdanakusuma D, Gayatri K. P. 2009. The relationship between blood type and the
formation of keloid post wound. Departemen / SMF Ilmu Bedah Plastik Fakultas Kedokteran
Universitas Airlangga, RSUD Dr. Soetomo, Surabaya.

Kischer CW, 1982. Fibronectin (FN) in hypertrophic scars and keloid. Cell and Tissue
Research 23 1(I): 29-37.

Savage K, Swann DA, 1985. A comparison of glycosaminoglycan synthesis by human
fibroblasts from normal skin, normal scar, and hypertropic scar. Journal Invest Dermatology
34(6) : 521-6.

Wolfram D,

Alexandar T.


Hypertrophic Scars and Keloids – A review of Their

Pathophysiology, Risk Factors, and Therapeutic Management. Available in: Dermatol Surg
2009; 35. 2009; 171-181

Ajab K., Shahzad M, Haroon T S. Keloids: Clinical Features and Management Part I.
Available in: Journal of Pakistan Association of Dermatologists 2006; 16: 97-103

Melinda S, Meites E, Daane SP. Keloids: Which Treatment is best for your patient?.
Available in: The Journal of Family Practice Vol 62 No 5. 2013: 227-233

Gauglitz Gerd G. Hypertrophic Scarring and Keloids: Pathomecanisms and Current and
Emerging Treatment Strategies. Available in: Mol Med 17 (1-2). 2011: 113-125

Universitas Sumatera Utara

Al-Attar A, Mess S, Thomassen JM . Keloid Pathogenesis and Treatment. Available in:
Plastic Reconstruction Surgery 117:286. 2006: 286-300


Seifert Oliver., Mrowietz Ulrich. Keloid Scarring: bench and bedside. Available in: Arch
Dermatol Res (2009) 301:259-272

Perdanakusuma DS, Noer MS, 2006. Penanganan parut hipertrofik dan keloid. Surabaya:
Airlangga University Press, pp. 4-18

Al-Attar, A, Mess, S, Thomassen, JM, Kauffman, CL & Davison, SP

2006, 'Keloid

pathogenesis and treatment', Plast Reconstr Surg, vol. 117, no. 1, pp. 286-300.

Sudigdo,F,Sofyan,I, 2008, Dasar – dasar metodologi penelitian klinis,ed. III. Sagung seto.

Gentur,S, 2011, Petunjuk praktis ilmu bedah plastik rekonstruksi, yayasan

khasanah

kebajikan. Sagung seto.


J. J. Brown and A. Bayat , ”Genetics susceptibility to raised dermal scarring,” British Journal
of Dermatology,vol.161, no.I, pp. 8-18,2004.

B. Shih and A. Bayat, “Genetic of keloid scarring,” Archives of Dermatological
Research,vol. 302,no. 5, pp. 319-339,2014.

A.G. Marneros, J. E. C. Norris,S. Watanabe,E. Reichenberger, and B. R. Olsen, “Genome
scans provide evidence for keloid susceptibility loci on chromosomes 2q23 and 7p11,”
Journal of Investigative Dermatology,vol. 122,no. 5, pp. 1126-1132,2004.

J. A. Clark,M. L. Turner,L. Howard,H. Stanescu,R. Kleta, and J. B. Kopp,”Description of
familial keloids in five pedigrees: evidence for autosomal dominant inheritance and
phenotypic heterogeneity,” BMC Dermatology, vol. 9, article 8, 2001.

K. M. Ramakrishnan, K. P. Thomas, and C. R. Sundararajan, “Study of 1,000 patients with
keloids in South India,,” Plastic and Reconstructive Surgery, vol. 53, no. 3, pp. 276-280,
1974.

Universitas Sumatera Utara


R. S. English and P. D. Shenefelt, “Keloid and hypertrophic scars,” Dermatologic Surgery,
vol. 25, no. 8, pp. 631-638, 1999.

Ishihara H, Yoshimoto H, Fujioka M (2000)

Keloid fibroblast resist ceramide-induced

apoptosis by overexxpression of insulin-like growth factor I receptor. J Invest Dermatol 115:
10655-1071.

Sayah DN, Soo C, Shaw WW (1999) Downregulation of apoptosis-related genes in keloid
tissues. J Surg Res 87 :209-216

Ghazizadeh M (2007) Essential role of IL-6 signaling pathway in keloid pathogenesis. J
Nippon Med Sch 7:11-22

Messadi DV, Doung HS, Zhang Q (2004) Activation of NfkappaB signal pathwayys in
keloid fibroblats. Arch Dermatol Res 296:125-133

Santucci M, Borbognoni L, Reali UM (2001) Keloids and hypertrophic scars of Caucasians

show distinctive morphologic and immunophenotypic profiles. Virchowws Arch 438:457463

Niessen FB, Schalkwwijk J, Vos H (2004) Hypertrophic scar formation is associated wwith
an increased number of epidermal Langerhans cells. J Pathol 202:121-129

Tuan TL, Wu H, Huang EY (2003) Increased plasminogen activator inhibitor-1 in keloid
fibroblast may account for their elevated collagen accumulation in fibrin gel cultures. Am J
Pathol 162:1579-1585

Zhang Q, Wu Y, Ann DK

(2003) Mechanisms of hypoxic regulation of plasminogen

activator inhibitor-1 gene expression in keloid fibroblasts. J Invest Dermatol 121:1005-1012

Universitas Sumatera Utara

Wu Y, Zhang Q, Ann DK (2004) Increased vascular endothelial growth factor may account
for elevated level of plasminogen activator inhibitor-1 via activating ERK1/2 in keloid
fibroblasts. Am J Physiol Cell Physiol 286:C905-C912


Chodon T, Sugihara T, Igawa HH (2000) Keloid-derived fibroblasts are refractory to Fasmediated apoptosis and neutralizzation of autocrine transforming growth factor-beta I can
abrogate this resiostance. Am J Pathol 157:1661-1669

Funayama E, Chodon T, Oyama (2003) Keratinocytes promote proliferation and inhibit
apoptosis of the underlying fibroblasts: an important role in tthe pathogenesis of keloid. J
Invest Dermatol 121:1326-1333

Lim IJ, Phan TT, Song C

(2001) Investigation of the influence of keloid-derived

keratinocytes on fibroblasts growth and proliferation in vitro. Plast Reconstr Surg 107:797808

Lee TY, Okochi H, Kim WJ (2004) Expression of transforming growth factor beta 1, 2, and
3 protein keloid s. Ann Plast Surg 43:179-184

Shaffer JJ, Taylor SC, Bodden F. Keloid scars: a review with a critical look at therapeutic
options. J Am Acad Dermatol. 2002:46(suppl):S63-S97


Atiyeh BS, Costagliola and Hayek (2005) Nonsurgical management of hypertropic scars:
evidence-base therapies, standart practices, and emerging methods. Aesthetic. Plast.Surg.
31:468-494
Datubo-Brown DD. Keloids: a review of the literature. Br J Plast Surg. 1990: 43:70-77

Rockwell WB, Cohen IK, Ehrlich HP. Keloid and hypertropic scars: a comprehensive
review. Plast Reconstr Surg. 1989:84:827-837

Universitas Sumatera Utara