MYCOBACTERIUM MYCOBACTERIUM
MYCOBACTERIUM MYCOBACTERIUM
THIS GENUS IS COMPOSED OF: Strictly aerobic, acid-fast rods, does not
Stain well gram stain indeterminant, DNA has high g+c content, unique cell wall,
Mycolic acid carbon chain length c60 Relatively slow growth two groups
A. RAPID GROWERS Visible colonies in 5 days
B. SLOW GROWERS Visible colonies in 5
days TYPE SPECIES:
Mycobacterium tuberculosis
THE GENUS MYCOBACTERIUM CAN BE DIVIDED INTO FOUR BROAD GROUPS
THE GENUS MYCOBACTERIUM CAN BE DIVIDED INTO FOUR BROAD GROUPS
1. THE TUBERCULOSIS COMPLEX
2. SLOW GROWING MYCOBACTERIA OTHER THAN TUBERCULOSIS MOTT
3. RAPIDLY GROWING MYCOBACTERIA
4. MYCOBACTERIUM LEPRAE
Acid Fastness Stain Acid Fastness Stain
Ziehl-Neelsen stain Ziehl-Neelsen stain
flood the slide with basic fuchsin a red dye in 5
phenol as a mordant.
heat gently for few minutes to melt the wax.
wash with 3 HCl in ethanol.
counter-stain with methylene blue.
Mycobacterium stains red and other bacteria and the background are blue.
The mycolic acid and its derivatives are responsible for the acid
f
THE TUBERCULOSIS COMPLEX THE TUBERCULOSIS COMPLEX
Organisms that resemble M. tuberculosis; Causing a similar type of disease in humans
1. M. tuberculosis 2. M. bovis
Mycobacterium tuberculosis Mycobacterium tuberculosis
General Features General Features
It is a causative agent for human tuberculosis.
It grows very slow with a generation time of
12-15 hours.
On solid media the colonies are raised and rough with a
wrinkled surface
.
M. tuberculosis cells grow either as discrete rods or as aggregates.
Virulent strains tend to grow as an aggregated long arrangement
called serpentine cord.
Cord factor
is a derivative of mycolic acids, trehalose 6-dimycolate.
Resistance Resistance
: :
UV Malachite
green1:13000 Alcohol to nonspore-
forming bacteria 3HCL, 6H
2
SO
4
, 4NaOH 15min
Heat62-63 ℃,15min
Chemical disinfectants more
Wet Dry highly
Sensitive Not sensitive
EUGONIC GROWTH 14 DAYS DYSGONIC GROWTH 14 DAYS
Mycobacterium tuberculosis Mycobacterium bovis
COLONIAL MORPHOLOGY OF THE TUBERCULOSIS COMPLEX MYCOBACTERIA
COLONIAL MORPHOLOGY OF THE COLONIAL MORPHOLOGY OF THE
TUBERCULOSIS COMPLEX MYCOBACTERIA TUBERCULOSIS COMPLEX MYCOBACTERIA
Transmission Transmission
Through respiratory tract, alimentary
tract, injured skin 。
TB in the lungs or throat can be infectious. This means that the bacteria can be spread
to other people. TB in other parts of the body, such as the kidney or spine, is usually
not infectious.
Who is at risk: Who is at risk:
Primary infection: children Secondary infection: age25
Virulence factors Virulence factors
No spore, no flagellum, no exotoxin,no endotoxin, no invasive enzyme
Capsule:
polysaccharide;CR3;enzyme; protect
LipidLipo arabinomannan Heat-shock proteinTuberculin
protein:
antigenicity, old tuberculin; associate with wax D can cause
hypersensitivity and form tubercle
Lipid: closely related to virulence a. Phospholipid
monocytes proliferate,cause tubercles
b. Wax D adjuventnot only to TB, delayed-type
hypersensitivity c. Sulfatide
硫酸脑苷脂
suppress phagosome combine with lysosome
d. Cord factor
trehalose-6,6-dimycolate destroy
mitochondria, cause chronic granulomatosis, suppress WBC wandering
Pathogenesis Pathogenesis
primary infection 1 lung infection
secondary infection
2 Out lung infection
Clinical syndromes Clinical syndromes
a. fatigue, weakness, weight loss and fever
b. pulmonary involvement: chronic cough,spit blood
c. meningitis or urinary tract involvement d. bloodstream dissemination: miliary
tuberculosis with lesions in many organs and a
high mortality rate .
Primary Tuberculosis Primary Tuberculosis
The organisms are transmitted among human via
aerosol.
TB bacilli lodge in the alveoli or lung alveolar ducts and
most of bacilli are phagocytosed by alveolar
macrophages.
Macrophages migrate to the hylar lymph node and
generate T cell-mediated immune response.
can be monitored by tuberculin test
Tuberculin Skin Test Tuberculin Skin Test
Tuberculin is a mixture known as purified protein derivatives PPD from TB bacilli.
It is a test for delayed type hypersensitivity. Positive reaction, reddening and thickening
5mm at the site of injection after
2-3 days
, indicates cellular immunity to tubercle bacilli.
Macrophages containing TB bacilli clump together and
begin to form tubercles. granulomatous response
With time, the centers of the tubercles become necrotic
and form cheesy acellular masses of caseous materials.
caseous lesion
Symptoms: Activation of macrophages - cytokine secretion, IL-1: fever,
TNF: lipid metabolism, weight loss, tissue necrosis. Oxygen radicals: tissue damages
Tissue necrosis - inflammation - mucous secretion, destruction of
blood vessels - frequent cough and bloody sputum
PULMONARY TUBERCULOSIS PULMONARY TUBERCULOSIS
Large caseating tubercle Miliary tubercles
HUMAN LUNG HUMAN LUNG
HUMAN LUNG HUMAN LUNG
TUBERCULOSIS TUBERCULOSIS
MYCOBACTERIUM TUBERCULOSIS MYCOBACTERIUM TUBERCULOSIS
Can infect disseminate and cause disease in many different body locations such as:
1. Meninges 2. Brain
3. Bone 4. Kidney
5. Essentially any organ lung primary target
Bacteria coughed up in sputum
Inhalation of bacteria
Bacteria reach lungs, enter macrophages
Bacteria reproduce in macrophages
Lesion begins to form caseous necrosis
Activated macrophages
Bacteria cease to grow; lesion calcifies
Immune suppression
Reactivation Lesion
liquefies
Dead phagocytes,
necrosis
M. tuberculosis