Penurunan Kadar Laktat Pada Pemberian Norepinefrin Dengan Plasebo Dan Norepinefrin Dengan Adjuvan Vasopresin Pada Pasien Syok Sepsis
DAFTAR PUSTAKA
Andersen, L. W., Mackenhauer, J., Roberts, J. C., Berg, K. M., Cocchi, M.
N., Donninq, M. W. 2013. Etiology and Therapeutic Approach to
Elevated Lactate Levels. Mayo Clinic 88(10) : 1127 – 1140.
Annane D, Bellissant E, Cavaillon JM, Mechanism of Septic Shock. The
Lancet. 2005;2:34-67.
Ballina M, Tracey Kj, Cholinergic Control of Inflamation. J Intern Med.
2009;265;663-679.
Barzegar, E., Ahmadi, A., Mousavi, S., Nouri, M., Mojtahedzadeh, M., The
Therapeutic Role of Vasopressin on Improving Lactate Clearance
During and After Vasogenic Shock: Microcirculation, is it The Black
Box. Acta Medica Iranica. 2016. 54(1) : 15 - 23.
Brooks GA, Dubouchad H, Brown M, Sicurello JP, Butz CE. 1999. Role of
mitochondrial lactate dehydrogenase and lactate oxidation in
intracellular lactate shuttle. Proc Nat Acad Sci USA, pp. 1129-34.
Butterworth IV JF, Mackey DC, Wasnick JD. Morgan & Mikhail’s clinical
anesthesiology, 5th Ed. United State of America: ; 2013; 825 – 842.
Chiolero R, Revelly JP, Tappy L, Energy metabolism in sepsis and injury.
Nutrition Jurnal. 1997, 13 (suppl 9):45S – 51S.
Cohen RD, Simpson R. 1975. Lactate metabolism. Anesthesiology.
Anesthesiology., pp. 43:661-73.
Connet RJ, Honig CR, Gayeski TE, Brooks GA. 1990. Defining hypoxia: a
system view of VO2, glycolysis, energetics, and intracellular pO2,
J. Appl. Physioi. pp. 68:833.
Daley MJ, Lat I, Mieure KD, Jennings HR, Hall JB, Kress JP. A
Comparison of Initial Monotherapy with Norepinephrine Versus
Vasopressin for Resuscitation in Septic Shock. Sage Journal. 2016.
Dellinger RP et al. Surviving Sepsis Campaign International Guideline for
Severe Sepsis and Septic Shock. Critical Care Med. 2013;41(2): 580637.
57
Universitas Sumatera Utara
58
Elamin M, Camporesi E, Evidence – based nutritional support in the
intensive care unit. International Anestesiology Clinics.2009;
47(1):121 – 138.
GA., Brooks. 2002. Lactate shuttles in nature. Biochem soc Trans. pp.
30:258-64.
Gunnerson, K. J., Saul, M. He S., Kellum, J. A. 2006. Lactate versus
nonlactate metabolic acidosis;a retrospective outcome evaluation of
critically ill patients. Critical Care;10
Guntur, A.H. Sepsis. Buku Ajar Penyakit Dalam FKUI. 2007; 2:1862-5.
Guyton, AC & Hall, JE. Pengaturan Keseimbangan Asam – Basa. Buku
Ajar Fisiologi Kedokteran (Ed.9). EGC. Jakarta. 1997.
Heyland DK et al, Should immunonutrition become routine in critically ill
patients.
Journal
American
of
anesthesiologist
medicine.
Blood
Analysis.
2001;286(8):944 – 53.
Higgins,
Article
Central
Venouse
Gases
AcuteCareTesting.Org,2011.
Hoffmann KF, Chappel D, Kammerer T, Jacob M, Paptistella M, Conzen P,
et all. Influence of an acetate and a lactate – based balanced
infusion solution on acid base physiology and hemodynamics. 2012;
17 – 21.
Homes TJ, Wiersinga, Poll TV, The host response to sepsis. Yearbook of
intensive Care and Emergency Medicine.2009;1;p39 – 48.
Hommes TJ, Wiersinga, Poll TV, The host response to sepsis. Yearbook of
Intensive Care and Emergency Medicine. 2009;1;p39-48.
Hood VI, Tannen RL. Protection of Acid – base Balance by pH Regulation
of Acid Production. NEJM 1998;12;819 – 25.
Leverve XM, Mustafa I. 2002. Lactate : a key metabolite in the intracellular
metabolic interplay. Crit Care, pp. 6:284-5.
Levy B, Gibot s, Franck, Cravoisy A, Bollaert PE. 2005. Relation between
muscle Na+K+ATPase activity and raised lactate concentration in
septic shock : a prospective study. [book auth.] Lancet, pp. 365:871.
Universitas Sumatera Utara
59
Lubis., 2006. Asidosis Laktat. Majalah Kedokteran Nusantara Volume 39.
No 1.
Malmir, J., Bolvardi, E., Aghae, M. A. 2014serum lactate is a useful
predictor of death in severe sepsis and septic shock. Reviews in
clinical Medicine, Vol 1.
Marini, J.J., Wheeler, A.P. 2006. Critical Care Medicine. Third Edition.
USA: Lippincott Williams & Wilkins
Marino PL, ICU BOOK, the 3rd Edition. Lippincot Williams & Wilkins.
2007; 254-60.
Martin GS, Mannino MA, Eaton S, Moss M, 2003. The Epidemiology of
Sepsis in the United States from 1970 through 2000. N Engl J Med.
2003;348;1546-1554.
Martin GS, Mannino MA, Eaton S, Moss,2003, The epidemiology of sepsis
in the united states from 1979 through 2000. N Engl J
Med.2003;348;1546-1554.
Meszaros K, Lang CH, Bagby GJ, Spitzer JJ. 1987. Contribution of
different organs to increased glucose consumption after endotoxin
administration. J. Biol. Chem, pp. 262: 10965-70.
Moviat M, van Haren F, van der Hoeven H. Conventional or
physicochemical approach in intensive care unit patients with
metabolic acidosis. [book auth.] Crit. Care. Crit. Care. s.l. : Crit.
Care., 2003, pp. R41-5.
Nguyen HB, Rivers EP, Knoblich BP, et al. Early Lactate Clearance Is
Associated With Improved Outcome in Severe Sepsis and Septic
Shock. Crit Care Med. 2004. Vol 32, No.8
Pasinato, et al. enternal nutritional Therapy in septic patients in the
intensive care unit: compliance with nutritional guide for critically
ill patients. Rev Bras Ter Intemsiva.2013;25(1):17
Presier JC, Metabolic response to the stress of critical illness. British
Journals Anesthesia,2014
Universitas Sumatera Utara
60
Rivers et al, Early goal- Directed Therapy In The Treatment of Severe
Sepsis And Septic Shock, the New England Journal of Medicine,
2001.
Russell JA, Walley KR, Singer J, Gordon AC, et al. Vasopressin versus
Norepinephrine Infusion in Patients with Septic Shock. The New
England Journal of Medicine. 2008. Vol. 358; No.9
Shapiro, Lactate Clearance vs Central Venouse Oxygen Saturation as Goals
of Early Sepsis metabolism. Ulusal Cer Derg2014;30;153-9.
Singer M. Deutschman C.S. Seymour C.W. et al. The Third International
Consensus Definitions for Sepsis and Septic Shock (Sepsis 3). JAMA.
2016;315: 801-810.
Tan J, Chen H, Chen X, Zhang D, He F. Vasopressin and Its Analog
Terlipressin versus Norepinephrine in the Treatment of Septic Shock
: a Meta-Analysis. Int J Clin Exp Med 2016; 9(7): 14183 - 14190
Valenza F, Aletti G, Fossali T, Chevallard G, Sacconi F, Irace M,
Gattinoni. Lactate as a marker of energy failure in critically ill
patients : hypothesis. [book auth.] Crit Care. Crit Care. s.l. : Crit
Care, 2005, pp. 9: 588-93.
Wasserman K, Mcllroy MB. 1964. Detecting the threshold of anaerobic
metabolism in cardiac patients during exercise. Am J Cardiol. pp.
14:844-52.
Weismuller K, Weigand MA, Hoffer S, The Neuroendocrine Axis : The
Nervous System and Inflammation. Annual Update in Intensive Care
and Emergency. 2012;3;12-16.
XM., Leverve. 1999. Lactic Acidosis. A New Insight? Minerva Anestesiol.
pp. 65:205-9
Zhou FH, Song Q. Clinical Trials Comparing Norepinephrine with
Vasopressin in Patient with Septic Shock: A Meta-Analysis. BioMed
Central. 2014.
Universitas Sumatera Utara
Andersen, L. W., Mackenhauer, J., Roberts, J. C., Berg, K. M., Cocchi, M.
N., Donninq, M. W. 2013. Etiology and Therapeutic Approach to
Elevated Lactate Levels. Mayo Clinic 88(10) : 1127 – 1140.
Annane D, Bellissant E, Cavaillon JM, Mechanism of Septic Shock. The
Lancet. 2005;2:34-67.
Ballina M, Tracey Kj, Cholinergic Control of Inflamation. J Intern Med.
2009;265;663-679.
Barzegar, E., Ahmadi, A., Mousavi, S., Nouri, M., Mojtahedzadeh, M., The
Therapeutic Role of Vasopressin on Improving Lactate Clearance
During and After Vasogenic Shock: Microcirculation, is it The Black
Box. Acta Medica Iranica. 2016. 54(1) : 15 - 23.
Brooks GA, Dubouchad H, Brown M, Sicurello JP, Butz CE. 1999. Role of
mitochondrial lactate dehydrogenase and lactate oxidation in
intracellular lactate shuttle. Proc Nat Acad Sci USA, pp. 1129-34.
Butterworth IV JF, Mackey DC, Wasnick JD. Morgan & Mikhail’s clinical
anesthesiology, 5th Ed. United State of America: ; 2013; 825 – 842.
Chiolero R, Revelly JP, Tappy L, Energy metabolism in sepsis and injury.
Nutrition Jurnal. 1997, 13 (suppl 9):45S – 51S.
Cohen RD, Simpson R. 1975. Lactate metabolism. Anesthesiology.
Anesthesiology., pp. 43:661-73.
Connet RJ, Honig CR, Gayeski TE, Brooks GA. 1990. Defining hypoxia: a
system view of VO2, glycolysis, energetics, and intracellular pO2,
J. Appl. Physioi. pp. 68:833.
Daley MJ, Lat I, Mieure KD, Jennings HR, Hall JB, Kress JP. A
Comparison of Initial Monotherapy with Norepinephrine Versus
Vasopressin for Resuscitation in Septic Shock. Sage Journal. 2016.
Dellinger RP et al. Surviving Sepsis Campaign International Guideline for
Severe Sepsis and Septic Shock. Critical Care Med. 2013;41(2): 580637.
57
Universitas Sumatera Utara
58
Elamin M, Camporesi E, Evidence – based nutritional support in the
intensive care unit. International Anestesiology Clinics.2009;
47(1):121 – 138.
GA., Brooks. 2002. Lactate shuttles in nature. Biochem soc Trans. pp.
30:258-64.
Gunnerson, K. J., Saul, M. He S., Kellum, J. A. 2006. Lactate versus
nonlactate metabolic acidosis;a retrospective outcome evaluation of
critically ill patients. Critical Care;10
Guntur, A.H. Sepsis. Buku Ajar Penyakit Dalam FKUI. 2007; 2:1862-5.
Guyton, AC & Hall, JE. Pengaturan Keseimbangan Asam – Basa. Buku
Ajar Fisiologi Kedokteran (Ed.9). EGC. Jakarta. 1997.
Heyland DK et al, Should immunonutrition become routine in critically ill
patients.
Journal
American
of
anesthesiologist
medicine.
Blood
Analysis.
2001;286(8):944 – 53.
Higgins,
Article
Central
Venouse
Gases
AcuteCareTesting.Org,2011.
Hoffmann KF, Chappel D, Kammerer T, Jacob M, Paptistella M, Conzen P,
et all. Influence of an acetate and a lactate – based balanced
infusion solution on acid base physiology and hemodynamics. 2012;
17 – 21.
Homes TJ, Wiersinga, Poll TV, The host response to sepsis. Yearbook of
intensive Care and Emergency Medicine.2009;1;p39 – 48.
Hommes TJ, Wiersinga, Poll TV, The host response to sepsis. Yearbook of
Intensive Care and Emergency Medicine. 2009;1;p39-48.
Hood VI, Tannen RL. Protection of Acid – base Balance by pH Regulation
of Acid Production. NEJM 1998;12;819 – 25.
Leverve XM, Mustafa I. 2002. Lactate : a key metabolite in the intracellular
metabolic interplay. Crit Care, pp. 6:284-5.
Levy B, Gibot s, Franck, Cravoisy A, Bollaert PE. 2005. Relation between
muscle Na+K+ATPase activity and raised lactate concentration in
septic shock : a prospective study. [book auth.] Lancet, pp. 365:871.
Universitas Sumatera Utara
59
Lubis., 2006. Asidosis Laktat. Majalah Kedokteran Nusantara Volume 39.
No 1.
Malmir, J., Bolvardi, E., Aghae, M. A. 2014serum lactate is a useful
predictor of death in severe sepsis and septic shock. Reviews in
clinical Medicine, Vol 1.
Marini, J.J., Wheeler, A.P. 2006. Critical Care Medicine. Third Edition.
USA: Lippincott Williams & Wilkins
Marino PL, ICU BOOK, the 3rd Edition. Lippincot Williams & Wilkins.
2007; 254-60.
Martin GS, Mannino MA, Eaton S, Moss M, 2003. The Epidemiology of
Sepsis in the United States from 1970 through 2000. N Engl J Med.
2003;348;1546-1554.
Martin GS, Mannino MA, Eaton S, Moss,2003, The epidemiology of sepsis
in the united states from 1979 through 2000. N Engl J
Med.2003;348;1546-1554.
Meszaros K, Lang CH, Bagby GJ, Spitzer JJ. 1987. Contribution of
different organs to increased glucose consumption after endotoxin
administration. J. Biol. Chem, pp. 262: 10965-70.
Moviat M, van Haren F, van der Hoeven H. Conventional or
physicochemical approach in intensive care unit patients with
metabolic acidosis. [book auth.] Crit. Care. Crit. Care. s.l. : Crit.
Care., 2003, pp. R41-5.
Nguyen HB, Rivers EP, Knoblich BP, et al. Early Lactate Clearance Is
Associated With Improved Outcome in Severe Sepsis and Septic
Shock. Crit Care Med. 2004. Vol 32, No.8
Pasinato, et al. enternal nutritional Therapy in septic patients in the
intensive care unit: compliance with nutritional guide for critically
ill patients. Rev Bras Ter Intemsiva.2013;25(1):17
Presier JC, Metabolic response to the stress of critical illness. British
Journals Anesthesia,2014
Universitas Sumatera Utara
60
Rivers et al, Early goal- Directed Therapy In The Treatment of Severe
Sepsis And Septic Shock, the New England Journal of Medicine,
2001.
Russell JA, Walley KR, Singer J, Gordon AC, et al. Vasopressin versus
Norepinephrine Infusion in Patients with Septic Shock. The New
England Journal of Medicine. 2008. Vol. 358; No.9
Shapiro, Lactate Clearance vs Central Venouse Oxygen Saturation as Goals
of Early Sepsis metabolism. Ulusal Cer Derg2014;30;153-9.
Singer M. Deutschman C.S. Seymour C.W. et al. The Third International
Consensus Definitions for Sepsis and Septic Shock (Sepsis 3). JAMA.
2016;315: 801-810.
Tan J, Chen H, Chen X, Zhang D, He F. Vasopressin and Its Analog
Terlipressin versus Norepinephrine in the Treatment of Septic Shock
: a Meta-Analysis. Int J Clin Exp Med 2016; 9(7): 14183 - 14190
Valenza F, Aletti G, Fossali T, Chevallard G, Sacconi F, Irace M,
Gattinoni. Lactate as a marker of energy failure in critically ill
patients : hypothesis. [book auth.] Crit Care. Crit Care. s.l. : Crit
Care, 2005, pp. 9: 588-93.
Wasserman K, Mcllroy MB. 1964. Detecting the threshold of anaerobic
metabolism in cardiac patients during exercise. Am J Cardiol. pp.
14:844-52.
Weismuller K, Weigand MA, Hoffer S, The Neuroendocrine Axis : The
Nervous System and Inflammation. Annual Update in Intensive Care
and Emergency. 2012;3;12-16.
XM., Leverve. 1999. Lactic Acidosis. A New Insight? Minerva Anestesiol.
pp. 65:205-9
Zhou FH, Song Q. Clinical Trials Comparing Norepinephrine with
Vasopressin in Patient with Septic Shock: A Meta-Analysis. BioMed
Central. 2014.
Universitas Sumatera Utara