MRSA Treatment in the Setting of Highly Active Antiretroviral Therapy HAART
Diagnosis and Treatment of Dengue Infection Dewi Dian Sukmawati
Division of Tropical and Infectious Disease Department of Internal Medicine
Udayana University School of Medicine-Sanglah Hospital Denpasar
Dengue infection, a mosquito – borne flavivirus, is caused by dengue virus
transmitted mainly by vector Aedes aegypti and Aedes albopictus, other species Aedes polynesiensis
and Aedes scutellaris play the role of less common vector. There are five serotypes, DEN-1, 2, 3, 4 and 5. Each episode of infection induces a life-long protective
immunity to the homologous serotype which confers partial and transient protection against subsequent infection by the other serotypes. Secondary infection by another
serotype is a major risk factor for DHF, mainly due to antibody induced enhancement. Epidemiologic studies have identified young age, female sex, high body-mass index, virus
strain or virulence and genetics of the human host e.g. major histocompatibility complex class I related sequence B and phospholipase C epsilon 1 genes as risk factors for severe
dengue. All five serotypes may be circulating in the population at any one time but from the experience in the south-east Asia it appears that the predominant circulating dengue
virus will show a sinusoidal pattern
– with a peak to peak interval of 7 – 9 years. It is likely that this interval allows a buildup of immune
– naïve population of children.
1,2,3,4
1. Course of Dengue Infection Infection by Dengue viruses may cause symptomatic infections or asymptomatic
seroconversion. Symptomatic dengue infection manifests as a systemic and dynamic disease with wide clinical spectrum. The spectrum ranges from non severe to severe
clinical manifestations. Following the incubation period, the illness begins abruptly and evolving in three phases: febrile, critical and recovery Figure 1. Due to its dynamic
nature, the severity of the disease will usually only be apparent around defervescence period during the transition of the febrile to the afebrile phase and often overlapping with
the onset of the critical phase.
5,6
Figure 1. The course of dengue fever
IgM = immunoglobulin M; IgG = immunoglobulin G. Temperature is given in degrees Celsius °C
Source: adapted from Yip, 1980 6