Malformations Possibly Caused by Smoking
achanceevent [86]
.Thedevelopmentofneuraltubedefectsisacontroversialtopic [97,98]
buttheassociationwithmaternalsmokingduringpregnancyislesslikely.
Theriskoforalcleftscleftlipandpalatehasbeenconsideredrepeatedlyinthecon- textofmaternalsmokingTable
8.5
[97,99] .Studiesrejectingsuchanassociationhave
occasionallyappeared:forexample,onerecentlypublishedstudyhasreportedhigherodds ratios1.09–1.85fororalcleftswithincreasingcigaretteconsumption,butnoassociations
wereidentiiedbetweencleftingtypeandsmokinghabits [100]
. Theassociationbetweenmaternalsmokingduringpregnancyandthedevelopmentof
cleftlipandpalateininfantshasbeendemonstratedelsewhere [97,99]
.Onecase-control studyconductedinMarylandbetween1992and1996revealednostatisticallysigniicant
associationbetweenmaternalsmokingandoralcleftdevelopment,althoughtherewasa slightincreaseintheC2alleleontheTGFagenotypeamongcasesoforalcleft
[46] .Incon-
trast,anotherUSstudyshowedincreasedrisksforisolatedcleftlipOR=2.1andforiso- lated cleft palate OR = 2.2 when the mothers smoked 20 or more cigarettesday. This
correspondstoa3-to11-foldriskincreasecomparedwithnon-smokers [101]
.Inastudy
Malformations Studypopulationn
RiskoroddsratioOR References
Malformations 86,000and288,067
livebirths Noassociationwithsmoking
OR0.98 [44,45]
Cleftpalatecleftlip 3-and11-foldincreasedrisk
[46,47] Microcephalus,cleft
defects,clubfoot 3,284livebirths+
4,500controls Associationconirmed
[48] Cleftlippalate
6.16-foldand8.69-foldrisk increaseinsmokers
[49] Cleftlippalate
1,002,742livebirths OR1.16cleftlip,1.29
cleftpalate [50]
Cleftlippalate Meta-analysis
1966–1996 OR1.29cleftlip+palate
and1.32cleftpalate [51]
Atrialseptaldefects Noassociationwithsmoking
[52] Downsyndrome
1,117,021livebirths Noassociationinmultiparous
women,butcannotbe ruledoutinprimiparous
women [53]
Limbdefectsandlimb reductiondefects
1,575,904and 1,109,299live
births610infants OR1.26–1.70
[54–56] Urinarytract
malformationsand polycystickidneys
118livebirths+369 controls,and
1,117,021live births
OR2.3and1.22respectively [57,58]
Aortopulmonary septaldefects
OR1.9 [56]
Craniosynostosis Increasedriskwithhigher
antenatalmaternal altitude
[59,60] Gastroschisis
Childrenbornto mothersbelow25
yearsofage OR2.0
[61]
Table 8.5
Malformationspossiblycausedbycigarettesmoking
conductedinalmost3.9millionlivebirthsintheUSA,2,207livebirthswithcleftlippalate wereidentiied
[102] .Asigniicantassociationwasfoundbetweenanyamountofmaternal
cigarette use during pregnancy and having a child with a cleft lippalate OR = 1.55. Additionalfactorssuchasmaternaleducationlevel,age,raceandmaternalmedicalcondi-
tionse.g.diabetesorpregnancy-associatedhypertensionwerepotentialconfounders.After adjustingfortheseconfounders,adose-responseassociationwithsmokingwasshown,by
comparisonwiththenon-smokingreferencegroup:theoddsratioswere1.501–10ciga- rettesday,1.5511–20cigarettesdayand1.7820cigarettesday
[102] .Infantscarrying
therarerC2alleleattheTaqIsiteintheTGFalocuswhowereexposedtomaternalsmoking weremorelikelytodevelopcleftpalate6.16-foldriskincreaseat10orfewercigarettesday,
8.69-foldriskincreaseatmorethan10cigarettesday [49,103]
. TGFa,TGFb3,RARAretinolicacidreceptorandtheproto-oncogeneBCL-3have
beenfoundtobeusefulmarkers,althoughallocationofthevariouscleftingtypestothe describedgenes,TGFa,TGFb3andMSX1,ispossiblypremature
[104] ,asisconjec-
turewhethermultivitaminjuiceconsumptionduringpregnancyinterfereswiththeTGFa gene
[105] .Anassociationbetweensmokingandtheoccurrenceofthisanomalyhasnot
beendemonstrated. Further malformations were identiied in a study conducted in four Polish districts:
theseoccurredinbetween1.1and1.9ofneonatesandmanifestedthemselvesprincipally aslimbreductiondefectsandfunctionaldisturbancesofstriatedmuscle
[106–108] .An
associationwithsmokingwaslesslikelythanwithharmfulenvironmentalfactors.Itis alsodebatablewhetherneuraltubedefectsaremorelikelytodevelopduringpregnancy
whenthemothersmokes [109]
.Seriousdefectsthathavealsobeenlinkedwithsmoking includechangesconsistentwithholoprosencephaly,aconditioncharacterisedbyabnormal
forebrainandmidfacialdevelopmentandbyfailureofcleavageintoleftandrighthemi- spheres. Compared with controls, maternal periconceptional exposures associated with
increasedrisksforholoprosencephalyincludedcigarettesmokingOR=4.1,andcom- binedalcoholandsmokingOR=5.4,andtheseincreasedrisklevelswereonlysurpassed
bywomenwithinsulin-dependentdiabetesOR=10.2 [110]
. Motherswhosmokeduringpregnancyaremorelikelytohaveoffspringwithcongeni-
talurinarytractanomaliesOR=2.3.ThisriskwashigherOR=3.7amonglightsmok- ers1–1,000cigarettesduringthepregnancythanamongheavysmokersOR=1.4
[57] .
Polycysticrenalchangeshavebeenreportedmostcommonly [58]
. Childbehaviourhasalsobeenstudiedintermsofmaternalsmokingduringpregnancy:
where the mothers were smokers, externalising behaviour problems aggressive, oppositional,overactiveweremoreprominentinthechildrenthaninternalisingbehaviour
problemsdepressed,anxious,withdrawn,andtheeffectofmaternalsmokingwasalmost identicalforboysandgirls
[111] .Accordingtoanalysesofleadlevelsintheblood,theirst
behaviouralproblemsinchildrenofsmokerscoincidewithraisedleadlevels [6]
.Smoking 10ormorecigarettesdayduringpregnancydoublestheriskOR=2.0ofinfantsbeing
non-babblersattheexaminationat8months [112]
.Thechild’scognitivedevelopmentis notaffectedbymaternalsmokingbehaviour;inthiscontext,socialbackgroundandlife-
stylearemoreimportantdeterminantsseealsoSect.7.1.4in Chap.7
, [113]
.According tootherestimates,prenatalexposureofthefoetustonicotinemayleadtobehavioural
disturbancesandcanindicatehigherriskforpsychiatricproblems,includingsubstance abuse
[114] .OneFrenchstudysuggeststhatbeingbornsmallforgestationalageatfull
termisassociatedwithpoorerschoolperformanceat12and18yearsandlaterentryinto secondaryschoolthanchildrenbornappropriateforgestationalageOR=2.3
[115] .