Effectiveness Out Patient Physical Exercise
Alcohol is also contains base. Besides, alcohol can
time and disappear during ten days.
also stimulate the secretion of gland so it increase the
hyper uricemia in the blood. Alcohol also produce much
hyper so make the special enzyme in the liver and
break the protein and 3 Third phase chronic
During this phase, the tofus crystal was formed.
This happened because of disobey the condition.
Usually it happened in 5- 6 times in a year. The
produce much hyper
pain will continue in uricemia. It is the same with
chemical drugs medicine. They can recover the disease
but also can be dangerous for vital organs in the body
such as kidney and liver. Safitri, 2012.
several joints, such as the edge of toe, the ankle,
knee, elbow, and the wrist became swelling.
4 Fourth phase tofus chronic
After ten years got this 3.
Patofisiology The first attack usually in
the edge of toe. It usually recover soon and the next
attack will come several years later. In the free phase, there
is no abnormal joints. That is why some people did not
realize that they got this disease, the patient will
get the swelling of hard crystal contain hyper
uricemia. The form was needle form in several
joints and auricles. The common joint which got
the attack is the joint of toes, ankle, knee, elbow,
disease. They think that they and fingers.
Dewani, got infection or something
else. The second and the third attack of hyper uricemia
came in the interval of months. It called the re sick
2006. d. Sign and symptom
It was serious pain, red, swollen, and feeling hot in
several joints. It happened in phase. The re sick
will the morning usually Dewani
recover often if it was chronic. The patient will feel
pain during all day. Dewani, 2006. The other theory stated
the sign and symptom of hyper uricemia namely :
2006. There were four phases of
hyper uricemia, namely: 1 First phase asimtomatik
During this phase, the increase of hyper uricemia
occurred without feeling pain and no crystal in
bladder. t 1
2
3 Pain, ache, and
uncomfortable surrounding the joints. The first attack
is in the toes joint. Swollen joints and the skin
was red purplish, erect, and cling, feeling warm,
and so sick when it touch. The joint was sick in cold
2 Second phase acute weather.
During this phase, the 4 Fever, chilling, and
joint got a big pain and uncomfortable
also the feeling hot. It was
emergency attack at night, so, the patient will wake
up from sleep. The attack will be serious in the short
1247 heart was beating faster.
This sign will be more serious for them whose
below 30 years old.
5
6
7 The first attack occurred in
special time, usually at night and in the morning
when wake up. The first attack was only
for one joint and occur during several days. It can
recover without the medicine. But in different
case, the attack can also in sole, knee, elbow, and
wrist. The sign will disappear
the content of bay leaves namely:
1 Flavonoid to make the blood circulation run
well and avoid the blood blockage, anti oxcidant
and analgetic.Utami 2013. Flavonoid in bay
leaves also avoid the hypertension, decrease
the hyper uricemia , and decrease blood glucose
Herliana, 2013
slowly, the joints can be 2 Tannin
is the active functioned normaly, and
no sign until the next attack.
material for anti inflammation and anti
microba Utami 2013. 8 The pain will appear again
if the patient consume base 3 Atsiri oil for analgesic
effect Utami 2013. high food
4 Alkaloids strong
9 If the pain will be massaged, it will make
more serious. So, make it analgetic to reduce pain
and anti inflammation Safitri 2012.
sure if you got the pain in this area. Know surely, it
is hyper uricemia or not. Getting the sure answer
whether it is hyper uricemia or not, it was
needed laboratories check up, radiological check up ,
and joints gland. Sutanto, 2013.
d. Bay leaves can be a
diuretic
so it can decrease the hyper uricemia in the
blood. Besides, it also can be the pain relief so it can be
used as the medicine of high blood pressure, hyper
uricemia, diabetes mellitus, gasitris, drunken of alcohol,
diarrhea, and wound Dewani, 2006.